high frequency for acne    


high frequency for acne

Myth #3: Acne is caused by stress The ordinary stress of day-to-day living is not an important factor in acne Severe stress that needs medical attention is sometimes treated with drugs that can cause acne as a side effect If you think you may have acne related to a drug prescribed for stress or depression, you should consult your physician

Sebum: The oily substance produced by sebaceous glands 
                                                                                  
Macule: A flat spot or patch of skin that is not the same color as the surrounding skin                                                                                     

Let’s take a look at some other popular beliefs about acne to dispel any other rumors; for example, stress. Is it true that increased stress spreads acne? Not exactly. What is true is that ultimately stress can have a very minimal or minor influence all by itself. It cannot actually cause acne, but it can influence reoccurrences of acne as stress increases the body’s creation of a substance called cortisol that in turn causes your sebaceous glands to produce increased amounts of sebum oil that can be blocked in pores. Note, however, that some medications people take when trying to deal with or treat stress can have a large influence on acne as the result of medication side effects.

The retinoids have great beneficial effects, but also some adverse effects, the main one being teratogenicity It is preferable not to use them in topical form for pregnant women, although a pregnancy test is only compulsory for tazaroteneOnly isotretinoin is used in systemic form It acts on all the factors of acne and offers long remissions, and sometimes complete cures Precautions must be taken for women of childbearing age due to its teratogenicity It is also important to be aware of its other adverse effects, explain them to the patient and, if possible, deal with them in advance

For a long time, the mantra of acne pathogenesis debates has been that acne vulgaris lesions develop when (supposedly largely androgen-mediated) increased sebum production, ductal hypercornification, and propionibacteria come together with local inflammatory process in the unlucky affected individual And yet, the exact sequence, precise interdependence, and choreography of pathogenic events in acne, especially the 'match that lights the fire' have remained surprisingly unclear, despite the venerable tradition of acne research over the past century

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